vestarВестник аритмологииJournal of Arrhythmology1561-86412658-7327НАО «Инкарт»vestar-561Research ArticleОБЗОРЫ ЛИТЕРАТУРЫ И МЕТА-АНАЛИЗЫREVIEWРОЛЬ КОННЕКСИНОВ И НАТРИЕВОГО КАНАЛА NAV1.5, КОДИРУЕМОГО ГЕНОМ SCN5A, В НАРУШЕНИЯХ ПРОВЕДЕНИЯ ЭЛЕКТРИЧЕСКОГО ИМПУЛЬСА В МИОКАРДЕROLE OF CONNEXINS AND SODIUM CHANNEL NAV1.5 CODED BY THE SCN5A GENE IN ALTERATIONS OF THE ELECTRIC IMPULSE CONDUCTION IN THE MYOCARDIUMПроничеваИ. В.PronichevaI. V.noemail@neicon.ruРевишвилиА. Ш.RevishviliA. Sh.noemail@neicon.ruНаучный центр сердечно-сосудистой хирургии им. А.Н.Бакулева РАМН, МоскваRussian Federation20122209202067675962Copyright © Проничева И.В., Ревишвили А.Ш., 20202020Проничева И.В., Ревишвили А.Ш.Pronicheva I.V., Revishvili A.S.Данная работа распространяется под лицензией Creative Commons Attribution 4.0.This work is licensed under a Creative Commons Attribution 4.0 License.https://vestar.elpub.ru/jour/article/view/561

Рассматривается роль межклеточных щелевых контактов в генезе нарушений ритма сердца, приводятся примеры мутаций, ассоциированных с фибрилляцией и асистолией предсердий.

The role of intercellular gap contacts in the pathogeny of cardiac arrhythmias are considered, examples of mutations associated with atrial fibrillation and atrial asystole are given.

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The authors declare that there are no conflicts of interest present.